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Lyme attacks the central nervous system
A bee sting procedure

Email from: "kay"
Date: Mon, 3 Jul 2000
Subject: [Lyme-Aid] BEE STINGS AND LYME


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Melittin is a small protein containing 26 a.a. residues and is the principal toxic component of the venom of the honey bee, Apis mellifera -50% of the honey bee venom is composed of melittin.

This toxic protein has been shown to integrate into natural or synthetic membranes and to disrupt a variety of cells and liposomes at micromolar concentrations.

In fact, when honey bee venom is injected through the sting into human organism, it causes lysis of the blood cells, depressing of the blood pressure and relesing of histamine, which in turn is responsible for the itching and the pain.

SAFETY DATA for melittin honey bee venom

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The Bee Informed, Summer 1999, Vol 6, #2 issue, page 7, states that melittin, a major ingredient (a 26 amino acid peptide) in honey bee venom "exhibited powerful in vitro inhibitory effects on the Lyme disease spirochete, Borrelia burgdorferi".

Clin Infect Dis 1997 Jul;25 Suppl 1:S48-51

"The antimicrobial agent melittin exhibits powerful in vitro inhibitory effects on the Lyme disease spirochete."

Lubke LL, Garon CF

Rocky Mountain Laboratories Microscopy Branch, National Institute of Allergy and Infectious Diseases, National Institutes of Health, Hamilton, Montana 59840, USA.

Borrelia burgdorferi has demonstrated a capacity to resist the in vitro effects of powerful eukaryotic and prokaryotic metabolic inhibitors.

However, treatment of laboratory cultures on Barbour-Stoenner-Kelly medium with melittin, a 26-amino acid peptide contained in honeybee venom, showed immediate and profound inhibitory effects when they were monitored by dark-field microscopy, field emission scanning electron microscopy, and optical density measurements.

Furthermore, at melittin concentrations as low as 100 microg/mL, virtually all spirochete motility ceased within seconds of inhibitor addition.

Ultrastructural examination of these spirochetes by scanning electron microscopy revealed obvious alterations in the surface envelope of the spirochetes.

The extraordinary sensitivity of B. burgdorferi to mellitin may provide both a research reagent useful in the study of selective permeability in microorganisms and important clues to the development of effective new drugs against Lyme disease.

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Email from: "kay"
Date: Sat, 8 Jul 2000
Subject: [Lyme-Aid] Dementia and Geriatric Cognitive Disorders

"Dementia and Geriatric Cognitive Disorders"

K. Kobayashia, C. Mizukoshia, T. Aokia, F. Muramoria, M. Hayashib, K. Miyazub, Y. Koshinoa, M. Ohtac, I. Nakanishic, N. Yamaguchia Department of Neuropsychiatry,

a Kanazawa University School of Medicine, b National Sanatorium Hokuriku Hospital, and c Department of Pathology, Kanazawa University School of Medicine, Kanazawa, Japan

"Borrelia burgdorferi-Seropositive Chronic Encephalomyelopathy: Lyme Neuroborreliosis?"

Dementia and Geriatric Cognitive Disorders 1997, 8:6:384-390.


A 36-year-old Japanese woman presented with progressive cerebellar signs and mental deterioration of subacute course after her return from the USA. Her serum antibody to spirochete Borrelia burgdorferi was significantly elevated.

A NECROPSY 4 years after her initial neurological signs revealed multifocal inflammatory change in the cerebral cortex, thalamus, superior colliculus, dentate nucleus, inferior olivary nucleus and spinal cord.

The lesions showed spongiform change, neuronal cell loss, astrocytosis and proliferation of activated microglial cells. The internal capsule was partially vacuolated and the spinal cord, notably at the thoracic level, was demyelinated and cavitated in the lateral funiculus. Microglial cells aggregated within and around the spongiform lesions and microglial nodules were present in the medulla oblongata.

Use of Warthin-Starry stain demonstrated silver-impregnated organisms strongly suggesting B. burgdorferi in the central nervous tissues.

The dentate nucleus and inferior olivary nucleus showed the most advanced lesions with profound fibrillary gliosis. Occlusive vascular change was relatively mild, and fibrous thickening of the leptomeninges with lymphocyte infiltrates was localized in the basal midbrain. The ataxic symptoms were due to the dentate and olivary nucleus lesions and mental deterioration was attributable to the cortical and thalamic lesions.

Spongiform change, neuronal cell loss, and microglial activation are characteristic pathological features in the present case. The cerebellar ataxia and subsequent mental deterioration are unusual clinical features of Lyme neuroborreliosis. Spirochete B. burgdorferi can cause focal inflammatory parenchymal change in the central nervous tissues and the present case may be an encephalitic form of Lyme neuroborreliosis.

For more information: Journals

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Source: Dr. Mercola's on-line alternative health newsletter, 7-23-00

Bee Venom Protocol For Lyme's Disease

Dr. Klinghardt uses 0.5 ml Bee-venom form Canada (Michael Simics) in 2.5 ml Procaine and injects 0.5 ml procaine in tender spot subcutaneously into the skin. It is relatively painless and has an incredible effect in his Lyme Disease patients. In some it lasts for 2 days and in others for 3-4 days. Dependent on the response he establishes a schedule: shots every 2-3 days. The client then learns to do their own injections and otherwise follow the bee venom protocol that we have (bee sting kit etc.). Patients start to feel much better very soon, their depression and fatigue lifts, then their pain. It appears to be a great benefit!

Glenroy Wolfsen ( wrote:

Some years ago when my Wife was still mobile and teaching school, (and I was her official aid) we began a special therapy to help her MULTIPLE SCLEROSIS. This was BEE VENOM Therapy. I got my bees from down south and my bee box from Maine and did the live bee stings as protocol indicated. (We were taught from a lady in middle New Jersey who was recognized then as doing some remarakable work with MS patients).

My wife and I noted IMMEDIATE improvement in sensation/warmth/ bladder control, pain relief and mental acquity as well as mobility. We continued the therapy for some time always with improvement. Because of her teaching schedule and the care of children at home (etc.) we were not as consistant with this therapy as we should have been - and because of that we cannot testify to more improvement as we did not continue (only because of our schedules).

Our neurologist was in favor of the home-treatment and had read positive literature on the subject. At that time live bees were the only way to get the most benefit of the venom - as laboratory venom was not as effective.

I therefore find this rather interesting and a positive finding. I will look forward to more reports on this new avenue for potential treatment of Lyme.

Glen Wolfsen (from beautiful New Jersey)

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